Lectures presented at the Opening Ceremony of the World Congress of Heart Disease meetings.
Since 2005, the Opening Lecture has been presented as the J.H.C Swan Memorial Lecture.
1989 | HEART FAILURE: ETIOLOGICAL MODELS
AND THERAPEUTIC IMPLICATIONS Edmund H. Sonnenblick, USA |
1993 | THE MOLECULAR BIOLOGY OF CARDIAC
HYPERTROPHY AND FAILURE: THE POTENTIAL FOR MOLECULAR INTERVENTION Robert Roberts, USA |
1995 | CARDIOMYOPATHY OF OVERLOAD: AN
UNNATURAL GROWTH RESPONSE IN THE FAILING
HEART Arnold M. Katz, USA |
1996 | PREVENTION OF HEART FAILURE -
FEASIBLE AND DESIRABLE Kanu Chatterjee, USA |
1997 | MORPHOLOGICAL AND FUNCTIONAL
TRANSFORMATION OF THE LEFT VENTRICLE IN
HEART FAILURE Sidney Goldstein, USA |
1998 | PREVENTION OF CORONARY DISEASE
EVENTS: MYTHS AND OPPORTUNITIES Harold J.C. Swan, USA |
1999 | NEW
CONCEPTS CONCERNING THE CELL BIOLOGY OF VULNERABLE PLAQUES James S. Forrester, USA |
2000 | THE CELLULAR AND MOLECULAR BASIS OF
HEART FAILURE IN VALVULAR REGURGITATION: RELATION TO CLINICAL OUTCOME Jeffrey S. Borer, USA |
2001 | MODERN
PERSPECTIVES ON DISEASES OF THE THORACIC AORTA John A. Elefteriades, USA |
2002 | GENETIC MECHANISMS OF HEART FAILURE Jeffrey A. Towbin, USA |
2003 | HEART FAILURE AND THE NEED FOR DEVICE THERAPY William T. Abraham, USA |
2005 | INTEGRATION OF CARDIAC MRI, CARDIAC CT SCANNING, AND NUCLEAR CARDIOLOGY IN CLINICAL ASSESSMENT OF PATIENTS WITH KNOWN OR SUSPECTED CHRONIC CAD: WHICH TEST IN WHICH PATIENT? Daniel S. Berman, USA |
2007 | HOW BEST TO IDENTIFY PATIENTS AT RISK FOR SUDDEN CARDIAC DEATH Michael E. Cain, USA |
2008 | WOMEN AND HEART DISEASE IN THE POST-WHI ERA Elizabeth L. Barrett-Connor, USA |
2010 | VOODOO DEATH REVISITED: THE MODERN LESSONS OF NEUROCARDIOLOGY Martin A. Samuels, USA |
2011 | ANGIOGENESIS IN ATHEROSCLEROSIS: MECHANISM, ROLE AND THERAPEUTIC IMPLICATION Jawahar L. Mehta , USA |
2012 | PERIPARTUM CARDIOMYOPATHY: NEW CONCEPTS IN
PATHOPHYSIOLOGY AND MANAGEMENT Uri Elkayam, USA |
2013 | CVD IN WOMEN: A FRESH PERSPECTIVE E.A. Amsterdam, USA |
2014 | GENDER DISPARITY IN CORONARY HEART DISEASE: BIAS, BIOLOGY, OR BOTH? |
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